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Site-specific arylation of rat glutathione S-transferase A1 and A2 by bromobenzene metabolites in vivo

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found that these two vectors were highly expressed in myocardial mitochondria and showed that inhibition of DIC and OGC increased mitochondrial ROS and GSH depletion, exacerbating ferroptosis in the myocardium [60]

Certain intervention studies have shown that treating with -T has a favorable effect on minimizing cancer risk when VE deficiency is present

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